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DOI: 10.1007/978-3-030-78672-4_6

A single molecular entity simultaneously activates both the GLP-1 receptor and the amylin receptor (calcitonin receptor complex), combining two complementary satiety and metabolic pathways: GLP-1 receptor activation : Glucose-dependent insulin secretion, glucagon suppression, delayed gastric emptying, and central appetite reduction through hypothalamic signaling Amylin receptor activation : Additional satiety signaling through the area postrema, complementary gastric emptying delay, and glucagon suppression through a distinct mechanism The unimolecular design means both receptor activations occur at a fixed ratio determined by the molecular structure

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It helps to reduce the tremor response in Parkinson's disease patients, reduces or eliminates tissue damage in diabetic patients, improves insulin resistance, reduces respiratory problems, reduces plaque build-up, and increases blood circulation in the arteries, among other effects