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According to previous investigations, APO ameliorated lung damage by reducing lipid peroxidation, inhibiting NOX expression and activity, blocking the NF-B pathway, and suppressing the transcription of pro-inflammatory cytokines in lung tissue (Kim et al

This suggests that while IL-4 can activate 12/15-LOX, the IL-4 concentrations in the lungs of asthma patients may not be sufficiently high to induce significant ALOX15 expression