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CTLA-4 CT60 (rs3087243) polymorphism and autoimmune thyroid diseases susceptibility: a comprehensive meta-analysis

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This creates a feedback loop that intensifies BBB disruption and neuroinflammation (Figure 3) [152,193,194]

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However, it is currently unclear whether individual organelles contribute to ferroptosis through impaired cell signalling, metabolism and biosynthesis of specific biomolecules or whether membrane lipids of these compartments are also direct substrates for iron-mediated oxidation that leads to cell death