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NRF2 activation induces NADH-reductive stress, providing a metabolic vulnerability in lung cancer

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drugs selected from the group consisting of an antioxidant, an antioxidant targeted to mitochondria, a histone deacetylase inhibitor, an inhibitor of mitochondria transition pore opening, an anti-inflammatory drug, a PPAR agonist, a RXR agonist, a sirtuin 1 agonist, an hypolipidemic drug and an autophagy activator

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The role of NLRP3 in estrogen-mediated alleviation of intestinal lymph fluid-induced lung injury following hemorrhagic shock