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AuNPs are known to mitigate neuronal apoptosis that is caused by glutamate toxicity, which is considered a key factor in neurodegenerative diseases

Furthermore, commonly employed proteasome inhibitors, like MG132, are designed to affect both the 20S and 26S proteasomes (Lee and Goldberg, 1998), posing challenges in specifically studying substrates degraded exclusively by the 20S proteasome

Figure 5 5 Relationship of ApoE with other neurodegenerative diseases Numerous studies have demonstrated the multifaceted impact of ApoE alleles on neurodegenerative diseases

In brain, the properties of GSH transport in isolated rat brain mitochondria seemed to be different from those reported previously for kidney mitochondria, as they were influenced most by inhibitors of the tricarboxylate carrier, citrate, isocitrate, and benzenyl-1,2,3-tricarboxylate (Wadey et al., 2009)

doi:10.4155/tde-2017-0056 Yousif H