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In addition, these genes were enriched in molecular functions such as antioxidant enzyme activity, phospholipase regulation, and peroxidase activity, suggesting their crucial roles in immune regulation and metabolic processes in RA

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This triad forms a pathogenic axis that drives disease progression, particularly in severe and treatment-resistant cases, and presents novel opportunities for therapeutic intervention ( Figure 4 )

TIM-3 further impairs T-cell function by binding to Galectin-9 (79)

The oxidative stress accompanying infections, the NADPH activation on macrophage plasma membranes, and the GSH consumption activate histone acetylation, and DNA unwinding that gives access to the extensive gene network that is NFKB dependent