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Xia W, Xiao J, Tong C, et al

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It was observed that the acrolein derivative FDP-lysine, an oxidative stress lipid oxidation end product, accumulated in RMCs of diabetic mice and downregulated Kir4.1 levels, while the acrolein scavenger 2-hydrazino-4,6-dimethylpyrimidine (2-HDP) prevented FDP-lysine accumulation and increased Kir4.1 expression in diabetic RMCs, and reversed diabetes-induced Kir channel deficiency in the perivascular region and RGC layer ( 4.2 Anti-VEGF therapy RMCs are involved in VEGF production and in the induction of neovascularization in DR