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As a high-affinity selective class IIa histone deacetylase (HDAC) inhibitor, TMP195 significantly alleviates tubular atrophy, interstitial fibrosis, and inflammatory responses in aristolochic acid nephropathy (AAN) mice by relieving G2/M phase arrest in TECs [326]

The condition sometimes indicates the presence of severe health issues including leukemia

Both animal [76] and human [77] studies suggest that VPA-induced hyperammonaemia may be enhanced via renal rather than hepatic mechanisms

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(ii) the TDP-dependent enzymes are destroyed and, parallel to their re-synthesis, a new requirement for their cofactors emerges