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Guo et al., 2016), they represent a promising disease-modifying therapeutic class

As described earlier, hyperactivation of these pathways due to increases in ROS levels may contribute to carcinogenesis

Although the use of primary brain endothelial cells and/or co-culture with other cells would provide a physiologically more relevant environment, bEND.3 cells consistently show a stable barrier function and thus have been widely used in previous studies on BBB permeability to therapeutics 35